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| Authors: | R.M. Cooper, D. Youle, A. Katerinas, C. Fox |
Abstract:
The interaction between E. amylovora and host cells can be divided into at least two distinct phases.
- Induction of bacterial genes encoding the expression of some determinant(s) of virulence, in response to a low molecular weight (<1500D), heat stable, choroform soluble compound of host cytoplasmic origin.
The elicitor is apparently a phenolic compound structurally related to chlorogenic acid and active at > 10-9M.
- A contact - dependent interaction between host and bacterial cells, resulting in the liberation of a diffusible, low molecular weight moiety displaying toxicity (directly or indirectly) to host cells.
Host activation of virulence gene(s) expression parallels that of Agrobacterium tumefaciens and Rhizobium spp; the elicitors are all phenolics but are distinct and uniquely active for each pathogen.
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